Athar, F. and Templeman, N.M. (2022). C. elegans as a model organism to study female reproductive health. Comparative Biochemistry and Physiology Part A: Molecular & Integrative Physiology 266: 111152.
Templeman, N.M., Cota, V., Keyes, W., Kaletsky, R., and Murphy, C.T. (2020). CREB non-autonomously controls reproductive aging through Hedgehog/Patched signaling. Developmental Cell 54(1): 92-105.
Botezelli, J.D., Overby, P., Lindo, L., Wang, S., Haïda, O., Lim, G.E., Templeman, N.M., Pauli, J.R., and Johnson, J.D. (2020). Adipose depot-specific upregulation of Ucp1 or mitochondrial oxidative complex proteins are early consequences of genetic insulin reduction in mice. American Journal of Physiology—Endocrinology and Metabolism 319: E529–E539.
Templeman, N.M., Luo, S., Kaletsky, R., Shi, C., Ashraf, J., Keyes, W., and Murphy, C.T. (2018). Insulin signaling regulates oocyte quality maintenance with age via cathepsin B activity. Current Biology 28(5): 753–760.
Templeman, N.M., and Murphy, C.T. (2018). Regulation of reproduction and longevity by nutrient-sensing pathways. Journal of Cell Biology 217(1): 93–106.
Templeman, N.M., Flibotte, S., Chik, J.H.L., Sinha, S., Lim, G.E., Foster, L.J., Nislow, C., and Johnson, J.D. (2017). Reduced circulating insulin enhances insulin sensitivity in old mice and extends lifespan. Cell Reports 20:451–463.
Templeman, N.M., Skovsø, S., Page, M.M., Lim, G.E., and Johnson, J.D. (2017). A causal role for hyperinsulinemia in obesity. Journal of Endocrinology 232(3): R173–R183.
Templeman, N.M., Mehran, A.E., and Johnson, J.D. (2016). Hyper-variability in circulating insulin, high fat feeding outcomes, and effects of reducing Ins2 dosage in male Ins1-null mice in a specific pathogen-free facility. PLOS ONE 11(4): e0153280.
Dionne, D.A., Skovsø, S., Templeman, N.M., Clee, S.M., and Johnson, J.D. (2016). Caloric restriction paradoxically increases adiposity in mice with genetically reduced insulin. Endocrinology 157(7): 2724-2734.
Templeman, N.M., Clee, S.M., and Johnson, J.D. (2015). Suppression of hyperinsulinemia in growing female mice provides long-term protection against obesity. Diabetologia 58(10): 2392-2402.
Templeman, N.M., Leblanc, S., Perry, S.F., and Currie, S. (2014). Linking physiological and cellular responses to thermal stress: β-adrenergic blockade reduces the heat shock response in fish. Journal of Comparative Physiology B 184(6): 719–728.
Mehran, A.E., Templeman, N.M., Brigidi, G.S., Lim, G.E., Chu, K.Y., Hu, X., Botezelli, J.D., Asadi, A., Hoffman, B.G., Kieffer, T.J., Bamji, S.X., Clee, S.M., and Johnson, J.D. (2012). Hyperinsulinemia drives diet-induced obesity independently of brain insulin production. Cell Metabolism 16(6): 723–737.
Templeman, N.M., Schutz, H., Garland, T., and McClelland, G.B. (2012). Do mice selectively bred for high locomotor activity have a greater reliance on lipids to power submaximal aerobic exercise? American Journal of Physiology: Regulatory, Integrative and Comparative Physiology 303(1): R101-R111.
Templeman, N.M., Beaudry, J.L., Le Moine, C.M.R., and McClelland, G.B. (2010). Chronic hypoxia- and cold-induced changes in cardiac enzyme and gene expression in CD-1 mice. Biochimica et Biophysica Acta 1800(12): 1248-1255.
Currie, S., Bagatto, B., DeMille, M., Learner, A., LeBlanc, D., Marks, C., Ong, K., Parker, J., Templeman, N.M., Tufts, B.L., and Wright, P.A. (2009). Metabolism, nitrogen excretion, and heat shock proteins in the central mudminnow (Umbra limi), a facultative air-breathing fish living in a variable environment. Canadian Journal of Zoology 88(1): 43-58.